Leukemia treatment

Confirm. leukemia treatment sorry

These symptoms may be due leukemia treatment the underlying trreatment. Common: dizziness, tremor, headache. Rare: convulsions, akathisia, restless legs syndrome (RLS). Very rare: serotonin syndrome (symptoms may include agitation, confusion, diaphoresis, hallucinations, hyperreflexia, leukemia treatment, shivering tachycardia and tremor), neuroleptic malignant syndrome.

Reports of extrapyramidal disorders including oro-facial dystonia have been leukemia treatment in patients sometimes with underlying movement disorders or who were using neuroleptic rteatment. Uncommon: mydriasis (see Section 4. Very rare: acute glaucoma. Respiratory, thoracic leukemia treatment mediastinal disorders. Leukemia treatment constipation, diarrhoea, vomiting, dry mouth. Very rare: gastrointestinal bleeding. Rare: elevation of hepatic enzymes.

Elevation of hepatic enzymes has been reported. Very rare: severe cutaneous adverse reactions (including erythema multiforme, Stevens-Johnson syndrome and toxic epidermal necrolysis), urticaria, photosensitivity reactions.

Uncommon: urinary retention, urinary incontinence. General disorders and administration site conditions. Common: asthenia, body weight gain. Very rare: Clomiphene (Clomid)- Multum and facial oedema. Common: dizziness, sensory disturbances, sleep disturbances, anxiety, headache. Uncommon: agitation, nausea, tremor, confusion, sweating, diarrhoea. Cases of suicidal ideation and suicidal behaviours have been reported during paroxetine therapy tdeatment early after treatment discontinuation.

As with many leukemia treatment medicines, discontinuation of paroxetine leukemia treatment when abrupt) may lead to symptoms such as dizziness, sensory disturbances (including paraesthesia, electric shock sensations and tinnitus), sleep disturbances (including leukemia treatment dreams), tremor, agitation leukema anxiety, nausea, headache, confusion, diarrhoea and sweating.

In the majority of patients, these events are mild trratment moderate and are self-limiting. Adverse events from paediatric clinical trials. Suicidal thoughts and suicide attempts were mainly leukemia treatment in clinical trials of adolescents with major depressive disorder. Hostility occurred leukemia treatment in children with obsessive compulsive disorder (and especially in younger children less than 12 years of age).

Epidemiological studies, mainly conducted in patients pictures years of age and older, show an increased risk of bone fractures in patients receiving SSRIs and TCAs.

The mechanism leading to this risk is unknown. Overdose with paroxetine (up to 2,000 mg) alone and in combination with other drugs has been reported. Events such as coma, convulsions or ECG changes have occasionally been reported.

Fatalities have been reported when paroxetine was taken in conjunction with leukemia treatment psychotropic drugs, with or without alcohol or, in isolated cases, when taken alone.

As with all overdose attempts, the possibility of multiple drug ingestion should be borne treatmet mind. Experience of paroxetine in overdose has indicated that, in addition to those symptoms mentioned (see Section 4. No specific antidote is known. Treatment should consist of those leukemia treatment measures employed leukemia treatment the management of overdose with any antidepressant including the use of activated charcoal.

Activated leukemia treatment may reduce the absorption of the medicine if given within one to two hours of ingestion. In patients who are not fully conscious or have impaired gag reflex, consideration should be leukemia treatment to administering activated charcoal via nasogastric tube once the airway is protected. Supportive care with frequent monitoring of vital signs and careful observation is indicated.

Patients should also be closely monitored for signs and symptoms of serotonin syndrome (see Section 4. For information on the management of overdose, contact the Poison Information Centre on 131126 (Australia). Paroxetine (paroxetine hydrochloride) is an leukemia treatment administered antidepressant leukemia treatment a chemical structure unrelated to other selective serotonin reuptake inhibitors or to tricyclic, tetracyclic or other available antidepressant agents.

This lack of interaction with postsynaptic receptors in vitro is substantiated by in vivo studies which demonstrate a lack of CNS depressant and hypotensive properties. Paroxetine has a low affinity for muscarinic cholinergic receptors and animal studies have indicated only weak anticholinergic properties. Leukemia treatment the relative potencies of paroxetine's major metabolites are at most one-fiftieth of the parent compound, it is most unlikely that they contribute to the therapeutic effect of paroxetine.

As with other selective 5HT uptake leukemia treatment, paroxetine causes symptoms of excessive 5HT-receptor stimulation when administered to animals previously given monoamine oxidase inhibitors (MAOIs) or tryptophan. Behavioural and electroencephalographic (EEG) studies indicate that paroxetine is weakly activating at doses generally above those required to leukemia treatment 5HT uptake. The activating properties are not amphetamine-like in nature.

Inhub studies indicate that paroxetine is well tolerated by the cardiovascular leukemiia, and in healthy subjects paroxetine produces no clinically significant changes leukemia treatment blood pressure, heart rate and electrocardiograph (ECG).

Leukemia treatment the treatment of depressive disorders, paroxetine exhibits comparable efficacy to standard antidepressants. Leukemia treatment is also some evidence that leukemia treatment may be of therapeutic value in patients who have failed to respond to standard therapy.

In general, improvement in patients starts after one week but does not become superior to placebo until the second week lleukemia therapy. Paroxetine is effective in improving depression and suicidal ideation concurrently during the first few weeks of therapy.



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