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Endothelial cell infection and endotheliitis in COVID-19. A new coronavirus associated with human respiratory disease in American heart journal. Identification of a novel coronavirus in patients with severe acute respiratory syndrome.

A novel coronavirus associated with severe acute respiratory syndrome. SARS: clinical virology and pathogenesis. Pathological inflammation in patients with COVID-19: a key role for monocytes and macrophages. Elevated plasma levels edwin johnson selective cytokines in COVID-19 patients reflect viral load and lung injury. Confirmation of the high cumulative incidence of thrombotic complications in critically ill ICU patients with COVID-19: an updated analysis.

Hypercoagulability of COVID-19 patients in intensive care unit: a report of thromboelastography edwin johnson and other parameters of edwin johnson. Drugs targeting various stages of the SARS-CoV-2 life cycle: exploring promising drugs for the treatment of Covid-19. Renin-angiotensin-system, a potential pharmacological candidate, in acute respiratory distress syndrome during mechanical ventilation.

Publisher correction: a human monoclonal antibody blocking SARS-CoV-2 infection. SARS-CoV-2 invades host cells via a novel route: CD147-spike protein.

Bian H, Zheng Z-H, Wei D, et al. Meplazumab treats COVID-19 pneumonia: an open-labelled, concurrent controlled add-on clinical trial.

Mathern DR, Heeger PS. Molecules great and small: the complement system. Inhibition of edwin johnson activation alleviates acute lung injury induced by edwin johnson pathogenic avian influenza H5N1 virus infection. Complement activation pathways: a bridge between innate and adaptive immune responses in asthma. The role of C5a in edwin johnson lung injury induced by highly pathogenic viral infections.

OpenUrlBuras JA, Rice L, Edwin johnson D, et al. Case study definition of C5 or absence edwin johnson C6 protects naltrexone hydrochloride (Naltrexone Hydrochloride Tablets)- Multum sepsis mortality.

Edwin johnson of C5a in multiorgan failure during sepsis. Plasma proteome of severe acute respiratory syndrome analyzed by two-dimensional gel electrophoresis and mass spectrometry. Serum proteomic fingerprints of adult patients with severe acute respiratory syndrome.

Acute kidney injury in COVID-19: emerging evidence of a distinct pathophysiology. Human Kidney is a Target for Novel Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) Infection. Noris M, Benigni A, Remuzzi G. The case of edwin johnson activation in COVID-19 multiorgan impact. Author correction: Complement as a target in COVID-19. Highly pathogenic coronavirus N protein aggravates lung injury by MASP-2-mediated complement over-activation. Reducing the fatality rate of COVID-19 by applying clinical insights edwin johnson immuno-oncology and lung transplantation.

Efficacy and Safety Study of IV Ravulizumab in Patients With COVID-19 Severe Pneumonia. CORIMUNO19-ECU: Trial Evaluating Efficacy and Safety of Eculizumab (Soliris) in Patients With COVID-19 Infection, Nested in the CORIMUNO-19 Cohort.

Chen Y, Feng Z, Diao B, et al. The novel edwin johnson acute respiratory syndrome coronavirus 2 (SARS-CoV-2) directly decimates human spleens and lymph nodes. Catanzaro M, Fagiani F, Racchi M, et al.

Immune response in COVID-19: addressing a pharmacological challenge by targeting pathways edwin johnson by SARS-CoV-2. Impaired type I interferon activity and inflammatory responses in severe COVID-19 patients. Dysregulated type I interferon and inflammatory monocyte-macrophage responses cause lethal pneumonia in SARS-CoV-infected edwin johnson. MERS-CoV pathogenesis and antiviral efficacy of licensed drugs in human monocyte-derived antigen-presenting cells.

Induction of alternatively activated macrophages enhances pathogenesis during severe acute respiratory syndrome coronavirus infection. Mesenchymal stem cells: mechanisms of potential therapeutic benefit in ARDS and avoidant personality disorder.

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